Recent evidence strongly suggests that an age-related decrease of vascular endothelial growth factor C (Vegfc) is causative to aging mice. In fact, activating vascular levels of Vegfc extends lifespan. Telomere shortening, genomic instability, epigenetic modifications, mitochondrial dysfunction, or loss of proteostasis also contribute to pathomechanisms. However, how the underlying genetic alterations condition the development of cardiovascular disease has remained largely unexplored.
This meeting at the University of Potsdam close to Berlin will bring together international leading experts, junior scientists and interested translational researchers that are dedicated to a better understanding of molecular and cellular processes during aging of the cardiovascular system.
This two-day symposium will cover molecular mechanisms of cardiovascular aging with an emphasis on:
- Age-related alterations in signal transduction
- Pathogenetic mechanisms of cardiovascular diseases
- Prevention of cardiovascular aging
- Disease models of cardiovascular aging
- Genetic traits of cardiovascular diseases and
- New therapeutic approaches
The Molecular Mechanisms of Cardiovascular Aging Symposium (MoCA) will be held on Thursday 16. till Friday 17. June 2022 as an in-person meeting.
Venue: Audimax, Neues Palais, University of Potsdam
Due-date for symposium contribution proposals: 09. May 2022
Notifications of acceptance: will be sent by 15. May 2022
Start of Registration: April 2022
Junior scientists are invited to attend the pre-satellite junior session on 15. June 2022. As places will be limited in this session, we recommend to register early.